Sodas, Pizzas and Other Junk Food May Put Kids at Liver Disease Risk

Sodas, Pizzas and Other Junk Food May Put Kids at Liver Disease Risk

Junk food is deliciously addictive, there’s no denying that fact. Munching on wafers, masala snacks, toffees and candies could make you happy, but don’t just fall for the taste trap. They contain nothing but empty calories, and can cause harm to your health. The shocking truth is also that these junk food come laced with artificial flavours, colours, preservatives and what not that you can’t be certain of what you are putting into your mouth. Foods like pizzas and burgers do contain veggies, meat and other nutritious ingredients but try and judge the downside – they are made with refined flour, processed cheese, sauces and dressings, that are highly calorific as well. While kids love these food items, it is important to make them understand the consequences of regular consumption of junk food. Junk food can lead to complications like obesity, chronic illness, low self-esteem and even depression, as well as affecting how they perform in school and extracurricular activities. In addition to this list, a recent study has found out that children who eat junk foods are more prone to liver malfunction.

Children who regularly intake fructose present in soda, sweetened beverages, pizza and salty food, biscuits, yogurt may be be prone to liver disease, researchers warn. According to a study, led by researchers from Bambino Gesu Hospital in Italy, dietary fructose increases serum uric acid concentrations. Both uric acid concentration and fructose consumption may be high in individuals with non-alcoholic fatty liver disease (NAFLD) – a condition where extra fat is accumulated in liver cells in people who drink little or no alcohol.

It is estimated to affect up to 30 per cent of the general population in Western countries and up to 9.6 per cent of all children and 38 per cent of obese children across a spectrum of liver disease, including NASH (defined as steatosis, hepatocyte ballooning and inflammation). Although NASH is a less aggressive form of NAFLD, it can progress to severe fibrosis and cirrhosis, with development of hepatocellular carcinoma in adults. The findings suggested that fructose consumption was independently associated with high uric acid, which occurred more frequently in patients with NASH than in not-NASH patients.


“It is plausible that dietary fructose intake and uric acid concentrations are potential risk factors for liver disease progression in NAFLD,” said Valerio Nobili from Bambino Gesu Hospital in Italy. “The study shows for the first time that uric acid concentrations and dietary fructose consumption are independently and positively associated with NASH,” Nobili added.

For the study, reported in the Journal of Hepatology, the team analysed 271 obese children and adolescents with NAFLD – 155 males, mean age 12.5 years – who underwent liver biopsy. Nearly 90 per cent were found drinking sodas and soft drinks one or more times a week. Almost 95 per cent of patients regularly consumed morning and afternoon snacks consisting of crackers, pizza and salty food, biscuits, yogurt, or other snacks. The development of NASH may markedly affect life expectancy and quality of life in affected individuals. Thus, “it is crucial to understand the risk factors for NASH in children and adolescents in order to design effective interventions,” Nobili said.

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